Your wait is over…
Your wait is over - Weight loss under a Medical Doctor’s supervision. Your weight will be over with a little effort . . . and then . . . Your life journey will start anew...
About Our Program
- Why this program?
- Did you know?
- Does the program require exercise?
- Are there lotions, potions, magic pills?
Possibly you have tried other weight loss programs previously and failed? Possibly you succeeded for a short time but then the weight came back? Maybe you tried once, twice or ten times, it doesn’t matter; today is a new day and a new beginning. This will absolutely be the most unique weight loss program you have ever experienced.
This program is holistic, scientific, rational, user friendly, and does not require calorie or carb counting. There are no foods, supplements, or vitamins to buy. You will not be required to eat food that is foreign to you. Also, there is no preset meal schedule, or rigid protocol to follow.
In the United States health insurance does not consider obesity a medical problem.
Instead it is considered cosmetic. In addition, since the percentage of obese people in the US is over 40% health insurance companies claim obesity to be a part of the ‘normal phenomena’ and therefore it does not call for a medical attention since 40% of people are obese.
Obesity is ‘normal’ according to most health insurance companies.
- Initial weight loss visit:
Starts for only $199.00 save $131.00
Value of $330.00
- Follow up visits:
Starts for only $140.00 save $40.00
Value of $180.00
- We offer a Package option:
For the price of $600, purchase 5 weight loss visits (value of $700).
Something to consider when deciding is: How much weight do you need to lose, and how rapidly do you want to lose it? At your initial appointment Doctor Jabaly will recommend what he thinks will work best for you based on the information you provide to him.
If you start paying per visit, and later decide to sign up for the package, the amount you already paid will be deducted from your package price.
Facts About Obesity
- Obesity and Poly Cystic Ovarian Syndrome:
- Genetics and Obesity:
- Central Obesity Versus Peripheral Obesity / Heart Disease:
- Obesity and it’s affect on blood pressure:
- Obesity and it’s affect on triglycerides (cholesterol):
- Obesity and it’s affect on diabetes:
- Epidemiology of Obesity:
- How can we measure obesity?
Many United States immigrants see a significant increase in their body weight approximately ten years after their arrival, if not sooner. This is largely attributed to the change from fresh, home cooked foods to more processed and or fast foods. Additionally, here in the US there is more eating when there is time to eat rather than when hungry. We also have more of a focus on heavy carbs such as breads, noodles, French fries, and baked potatoes rather than fresh vegetables, and salads. Also, we Americans tend to be more sedentary than the rest of the world. Certainly you’ve heard the line about fast foods and sedentary lifestyle until you are blue in the face, but were you aware that immigrants from nearly ANY OTHER COUNTRY experience this weight gain? That certainly makes a sad statement about our eating, health, and nutrition as Americans.
Ghrelin is a peptide that is produced in the stomach and the duodenum that stimulates eating. Leptin is a protein that is produced in adipose tissue, and it provides negative feedback to appetite-control centers. Central nervous system neurotransmitters are involved in appetite control, and they may be modulated with medication.
Antidepressants, antipsychotics, anticonvulsants, and hypoglycemic agents can all contribute to weight gain.
Metabolic syndrome increases the risk of type 2 diabetes, coronary artery disease, and cerebrovascular disease.
Smoking is the largest avoidable cause of cancer, and obesity is the second largest. An increased risk of death from several cancers is seen among obese patients, including esophageal, colon, kidney, gallbladder, and pancreatic cancer as well as non-Hodgkin’s lymphoma and multiple myeloma; there is also a trend toward an increased risk of prostate, gastric, ovarian, and endometrial cancer. Breast cancer has been most closely associated with central obesity.
Being overweight is a major risk factor for Obstructive Sleep Apnea.
Fatty liver disease is the most common cause of elevations in liver enzymes, and it affects 20% of the U.S. population.
The association between obesity and degenerative joint disease among adults is most often seen in weight-bearing joints such as the hips and knees.
The U.S. Preventative Services Task Force recommends screening for obesity in adults; this can be done with three simple steps: measuring BMI, measuring waist circumference, and identifying comorbidities.
In a study that compared four different popular diets, all were equally effective with regard to resultant weight loss, but at 1 year the maintained weight loss was only 2 Lbs.
Low- carbohydrate diets may be difficult to adhere to long term, and they may adversely affect nutritional status.
Very-low-calorie diets may produce more rapid initial weight loss, but they produce similar results as other diets over time. A low-calorie diet is most often employed. When the patient chooses less energy-dense food, the volume of food eaten may stay about the same.
To qualify for bariatric surgery, a patient should have a BMI of more than 40 and weight-related complications.
With rates rising to unprecedented levels with each passing year, obesity is rightly referred to as an epidemic and is, arguably, the most poorly controlled and potentially dire health threat facing the United States. Obesity is the major modifiable risk factor for type 2 diabetes (itself now epidemic) and a major contributor to most predominant causes of premature death and disability, including but not limited to cardiovascular disease, cancer, stroke, obstructive pulmonary disease, and degenerative arthritis. Secular trends are similar in most other developed countries. Cultural transitions in developing countries are associated with a rapid rise in the rate of obesity as well, even while historical public health scourges such as microbial diseases persist. Obesity thus constitutes a global health crisis. At the 10th International Congress on Obesity in Sydney, Australia, in September 2006, it was announced that for the first time in history, the planet-wide population of overweight (over 1 billion) outnumbered the hungry (roughly 600 to 700 million).
An inability to curtail the spread of obesity has resulted in a tendency to exaggerate the complexities, if not the difficulties, of the challenges involved. While the investigation of obesity rightly subsumes metabolism, genetics, lipidology, endocrinology, and even newly emerging disciplines such as nutrigenomics, it must be conceded that human physiology is much the same as it ever was and thus cannot house the explanation for suddenly skyrocketing obesity rates. That answer resides in an environment that is not the same as it was before, rendering human adaptations to a world of caloric scarcity and a high demand for physical exertions largely obsolete. In short, our patients (and we) are getting fat in record numbers for the simple reason that they (and we) can do so- for the first time in history. It is scarcely an exaggeration to say that human intelligence, since it first evolved, has been dedicated to making obesity possible by establishing a reliable supply of palatable food and by inventing technologies to reduce the physical ardors required for survival. We have become, in the fullness of time, victims of our own resourcefulness and success.
But while accounting for the obesity epidemic is simple, reversing it will be anything but easy-and from this derives the false and even harmful perspective that complexity impedes our progress. This view is false because a reduction in caloric intake to the level required for maintenance of a healthful weight is all that is universally needed for weight control. This view is harmful because it shifts attention and resources from the demanding but potentially rewarding task of pursuing what we know, to the often-frivolous task of parsing what we do not.
The role of clinicians in contending with this challenge is itself subject of debate. The US Preventive Services Task Force has expressed ambivalence about the utility of clinical counseling for weight control. We are accustomed in medical research to some degree of reductionism, the study of active ingredients. Thus, when obesity interventions are studied, they are generally examined discretely, independently of societal trends. When such interventions fail to make appreciable differences in the outcome(s) of interest- generally some measure of weight-we conclude that they are ineffective. Or, at best, we fail to conclude that they are effective.
But the “mass” against which we are working is daunting. The world is powerfully, and ever more, obesigenic. Even interactions that apply an effective counterforce may fail to move this massive and ever-accumulating resistance. The implications are that for there to be any hope of curtailing the obesity epidemic, we must apply all reasonable countermeasures concurrently..
Despite its limitations when applied to an individual, BMI performs well at the population level for several reasons. BMI trends reflect trends in adiposity, not muscularity. There is nothing to suggest that increasing legions of the muscular and fit are responsible for consistent increases in BMI in the United States and other countries; there is much to suggest that rising BMI is indicative of increasing adiposity. The distinction between excess body fat and muscularity is easily made at the individual level, and thus the use of the BMI is unlikely to generate clinically relevant confusion. Finally, such crude measures as the BMI, and even casual inspection, correlate fairly well with costly and sophisticated measures of adiposity.
Overweight in adults is defined as BMI at or above 25kg per m2.
Adult obesity is defined in stages.
Stage 1 obesity is a BMI of 30 to 34.9. Stage 2 obesity is a BMI of 35 to 39.9; and. Stage 3 obesity is a BMI of 40 or higher.
A BMI of 25 to 29.9 is “overweight.”
Stage 3 obesity was formerly known as “morbid obesity.” Current Definitions of Overweight and obesity in Adults. BMI
Category < 18 Underweight 18 to < 25 Healthy Weight 25 to < 30 Overweight 30 to < 35 Stage I Obesity 35 to < 40 Stage II Obesity > 40 Stage III Obesity (formerly “morbid” obesity)
The name change is appropriate and important for two reasons.
First, although a BMI of 40 is quite high, it is not invariably associated with morbidity.
Second, and of greater importance epidemiologically, morbidity is often induced by obesity at a BMI well below 40. The risks of complications of excess adiposity may, in general, be considered low, moderate, and high as BMI rises through overweight to stage III obesity, but the actual risk in an individual will vary.